How to Find a Menopause Specialist: What to Look For and Ask
The timing of hormone therapy matters as much as the hormones themselves: starting within ten years of menopause produces different outcomes than starting a decade later.
The Menopause Society Certified Menopause Practitioner (MSCP) credential is the most reliable publicly available filter for genuine specialist expertise.
Micronized progesterone and 17-beta estradiol are bioidentical, FDA-approved, and the current evidence-based standard of care for most women on hormone therapy.
Testosterone is the most abundant sex hormone in premenopausal women and its decline deserves clinical attention, not just the estrogen story.
A normal FSH during perimenopause does not rule out the transition: perimenopause is a clinical diagnosis, not a laboratory one.
Telehealth has made specialist-level menopause care accessible to women who previously had none, with the same clinical standards and evidence base as in-person programs.
Baseline lab testing before prescribing is non-negotiable: a provider who skips it is not individualizing care.
Searching "menopause specialist near me" is one of the most common health queries among women in their forties and fifties, yet the results are often a confusing mix of gynecologists, naturopaths, functional medicine clinics, and weight loss centers, each claiming expertise in hormonal health. The search itself reveals a problem that goes deeper than geography: there is no single, universally recognized credential for menopause medicine, which means the quality of care varies enormously from one provider to the next. For a woman experiencing vasomotor symptoms, cognitive changes, sleep disruption, and accelerating cardiovascular risk, finding the right clinician is not a matter of convenience. It is a matter of how well the next several decades of her life will unfold.
The science of menopause has undergone a genuine reckoning over the past two decades. The 2002 Women's Health Initiative (WHI) study caused a sharp drop in hormone therapy prescriptions after it suggested elevated risks of breast cancer and cardiovascular disease, and millions of women were left to manage symptoms without medical support. Subsequent reanalysis of that data, along with a body of newer research, has substantially revised that picture, demonstrating that the timing of hormone therapy initiation, the type of hormones used, and a woman's individual risk profile all determine outcomes in ways the original WHI design could not capture [1]. The result is a field in which the evidence has moved faster than clinical practice, and finding a specialist who has kept pace with that evidence is now the central challenge for millions of women.
The quality of menopause care depends less on a clinician's specialty than on their engagement with current evidence, their willingness to individualize treatment, and their ability to situate hormonal health within the broader context of aging.
Why Menopause Requires Specialist-Level Thinking
Menopause is not a disease, but it is a profound physiological transition with measurable downstream effects on multiple organ systems. Estradiol, the dominant form of estrogen during reproductive years, functions less like a female sex hormone and more like a systemic maintenance signal. Receptors for estradiol are found in the brain, heart, bone, skeletal muscle, skin, gut, and immune tissue, which is why its withdrawal triggers symptoms that span far beyond the reproductive system [2]. The hot flush is simply the most visible symptom of a much broader biological reorganization.
Consider what happens at the level of the cardiovascular system. Before menopause, estradiol supports endothelial function, the capacity of blood vessel linings to dilate and regulate blood flow, and suppresses inflammatory signaling in arterial walls. After menopause, LDL cholesterol rises, HDL function changes, and arterial stiffness accelerates. The ten-year cardiovascular risk gap between men and women, which previously favored women, closes rapidly in the postmenopausal years [3]. A clinician who frames menopause solely as a symptom management problem will miss the preventive cardiovascular window that evidence increasingly suggests is open in the early postmenopausal years [1].
Bone is similarly affected. Peak bone density is established in the late twenties, and estrogen is the principal brake on osteoclast activity, the cellular process that breaks bone down. In the first five years after menopause, women can lose three to five percent of bone density per year, a rate that slows but never fully stops [4]. Then there is the brain. Estradiol supports glucose metabolism in neurons, modulates acetylcholine and serotonin signaling, and reduces amyloid accumulation. Observational data consistently link the menopausal transition to changes in verbal memory and processing speed, and emerging research suggests that early initiation of hormone therapy may influence long-term cognitive trajectories [5].
A generalist physician treating menopause as a short-term symptom management problem will calibrate care around the next six months. A genuine menopause specialist will calibrate it around the next thirty years.
What Makes Someone a Menopause Specialist
The term "menopause specialist" is not a protected medical title in most countries. Any physician can self-designate as one. This reality makes credential verification more important, not less. The most meaningful marker of genuine expertise in North America is the Menopause Society Certified Menopause Practitioner (MSCP) credential, awarded by the Menopause Society (formerly the North American Menopause Society, NAMS). Practitioners who hold this credential have completed specific training in menopause medicine and passed a standardized examination covering hormonal physiology, clinical management, and risk-benefit assessment [6]. It is an imperfect filter, but it is the most reliable publicly available one.
Beyond certification, specialty background matters in a nuanced way. Gynecologists, endocrinologists, and internal medicine physicians all have legitimate pathways to menopause expertise, but none of their base training guarantees it. A gynecologist may be exceptional at surgical management of fibroids but have given menopause medicine minimal attention since residency. Conversely, a family medicine physician who has dedicated continuing medical education hours to hormonal health may provide more sophisticated care than a gynecologist who reflexively refuses all hormone prescriptions based on a 2002 reading of the WHI data. The credential and the demonstrated clinical philosophy matter more than the specialty label on the door.
Fellowship training through the Menopause Society, active membership in the British Menopause Society or the International Menopause Society, and recent publications or conference presentations in hormonal health are all signals worth examining. Clinicians who teach what they practice tend to stay current, because teaching requires updating. A provider who can cite the Kronos Early Estrogen Prevention Study (KEEPS), the ELITE trial, or the nuances of the "timing hypothesis" demonstrates that their knowledge is not frozen in the early 2000s [1] [7].
The Timing Hypothesis: Why It Changes Everything
The most important conceptual shift in menopause medicine over the past two decades is the timing hypothesis, sometimes called the "window of opportunity" for hormone therapy. The original WHI study enrolled women with a mean age of 63, most of whom were more than ten years past their final menstrual period. When harm signals appeared in that population, clinicians incorrectly generalized those findings to all postmenopausal women, including those who were recently menopausal [1].
Subsequent analyses, including the WHI's own age-stratified data and the ELITE trial, which compared early versus late initiation of estradiol on carotid intima-media thickness, a structural marker of atherosclerosis, found that women who began hormone therapy within ten years of menopause showed cardiovascular benefit rather than harm [7]. The biology is not mysterious. When arteries have been in an estrogen-rich environment for decades, estradiol receptors on endothelial cells remain functional and responsive. When those same arteries have spent a decade in an estrogen-depleted state and atherosclerotic plaques have already formed, re-introducing estrogen into an inflamed environment produces different, less predictable effects. The vessel's receptor landscape has changed.
A clinician who understands the timing hypothesis will ask not "should you take hormones?" but "how long has it been since your last period, and what does your cardiovascular risk profile look like right now?"
This single conceptual shift transforms the clinical conversation. It means that a 51-year-old woman who began experiencing perimenopausal symptoms a year ago is in a different risk-benefit position than a 65-year-old woman ten years postmenopausal, even if their symptom burden is identical. Any clinician who applies identical caution to both, or who refuses hormone therapy discussion entirely based on a blanket reading of the WHI, is not practicing evidence-based medicine. Finding a specialist means finding someone who reasons from the timing hypothesis, not around it.
Bioidentical vs. Synthetic Hormones: Separating Science from Marketing
One of the most reliably confusing areas of menopause medicine is the debate over "bioidentical" hormones, a term that has been co-opted by both legitimate clinical practice and unregulated compounding pharmacy marketing. The confusion is worth unpacking carefully, because a well-informed patient navigates it more effectively than one who arrives with fixed opinions in either direction.
Bioidentical hormones are molecules that are chemically identical to the hormones the human body produces. The key ones in menopause medicine are 17-beta estradiol (the dominant form of estrogen in premenopausal women) and micronized progesterone (a form of progesterone molecularly identical to endogenous progesterone). Both are available as FDA-approved medications: estradiol as patches, gels, and sprays; micronized progesterone as oral capsules and vaginal formulations. These are not fringe products. They are the standard of care recommended by the Menopause Society and major international bodies [8].
The distinction that matters clinically is not between "bioidentical" and "synthetic" as marketing categories, but between FDA-approved formulations and compounded preparations. Compounding pharmacies prepare custom hormone mixtures outside the standard regulatory approval process, which means their potency, purity, and absorption characteristics are less consistently verified. Some compounded preparations, particularly those containing estriol or combinations like Bi-Est (a blend of estradiol and estriol), are used by experienced clinicians in specific contexts, but the evidence base for many compounded regimens is substantially thinner than for approved formulations [8].
Synthetic progestins, like medroxyprogesterone acetate (the progestin used in the original WHI), differ meaningfully from micronized progesterone in their receptor binding profiles and downstream effects on lipids and breast tissue. Multiple analyses suggest that micronized progesterone carries a more favorable cardiovascular and breast cancer risk profile than synthetic progestins, which is why major guidelines now recommend it preferentially for women with a uterus who require progestogen protection [9]. A menopause specialist knows this distinction and can explain the clinical rationale for the formulation they prescribe. A provider who conflates all progestogens, or dismisses the distinction as irrelevant, is not operating at specialist level.
For women seeking hormone therapy, products like Estradiol Patch, Micronized Progesterone, and Bi-Est 50/50 Cream represent the spectrum of evidence-based formulations available through specialized hormone care programs. The right formulation depends on individual physiology, symptom pattern, route of administration preferences, and a thorough baseline laboratory assessment.
The Role of Testosterone in Female Hormonal Health
Testosterone is rarely discussed in the context of women's health, yet it is the most abundant sex hormone in premenopausal women by absolute concentration. Ovarian and adrenal production of testosterone begins declining in the late thirties, a process that accelerates during the menopausal transition. By the time a woman is fully postmenopausal, testosterone levels may be fifty to seventy percent lower than they were in her mid-twenties [10].
The clinical consequences of this decline extend beyond libido, though hypoactive sexual desire disorder is the most studied and the only FDA-acknowledged indication for testosterone in women. Low testosterone in women is also associated with fatigue, reduced motivation, difficulty with body composition, and diminished muscle anabolism. Given that sarcopenia, the progressive loss of skeletal muscle mass and strength, is one of the strongest predictors of disability and mortality in aging, any intervention that supports muscle protein synthesis and mitochondrial function in muscle tissue deserves serious consideration [10].
The evidence base for testosterone in women is limited by a near-complete absence of FDA-approved female formulations in most markets, which has historically meant that women receive off-label prescriptions of male formulations at lower doses. The Global Consensus Position Statement on the Use of Testosterone Therapy for Women, published in 2019 across multiple major endocrinology and menopause journals, concluded that evidence supports testosterone use in postmenopausal women for hypoactive sexual desire disorder, with no serious adverse events at physiological doses [10]. A menopause specialist will discuss testosterone as a legitimate component of hormonal assessment, not an afterthought. Formulations like Testosterone Topical Cream and Testosterone Gel are used in female hormone health programs at doses calibrated for female physiology.
Baseline Testing: What a Thorough Assessment Looks Like
Before initiating any hormone therapy, a rigorous specialist will conduct a baseline assessment that goes well beyond a single estradiol measurement. Hormonal health exists within a metabolic, cardiovascular, and inflammatory context, and optimizing it requires understanding that context. A complete female hormone panel should include FSH (follicle-stimulating hormone), LH, estradiol, total and free testosterone, sex hormone-binding globulin (SHBG), DHEA-S, and progesterone, interpreted together rather than in isolation. SHBG levels, for instance, determine how much testosterone is biologically available to tissues, and high SHBG driven by oral estrogen can paradoxically leave a woman with adequate total testosterone but minimal free testosterone [10].
Cardiovascular risk markers, thyroid function, fasting glucose, and insulin levels round out a baseline that allows a clinician to prescribe hormones with genuine individualization rather than by algorithm. A Complete Female Hormone Panel provides the full hormonal picture needed to make these decisions with precision. The Menopause Society's clinical guidelines recommend individualized risk stratification before prescribing, accounting for personal and family history of cardiovascular disease, venous thromboembolism, breast cancer, and bone density [8].
A clinician who initiates hormone therapy without baseline laboratory work, or who offers a single symptom-driven prescription without discussing the broader hormonal picture, is not practicing at specialist level. The baseline assessment is where individualization begins, and individualization is precisely what distinguishes menopause medicine from one-size-fits-all prescribing.
Questions to Ask a Prospective Menopause Specialist
Walking into a first consultation with the right questions is one of the most effective ways to assess whether a clinician is genuinely expert in this area. The goal is not to interrogate but to understand the clinical philosophy behind the care being offered. A specialist confident in their knowledge will welcome these questions. A provider who becomes defensive or dismissive when asked about evidence is revealing something important about how they practice.
Ask how many patients the clinician currently manages on hormone therapy, and what their approach is to the timing of initiation. A high-volume menopause practice means the provider has seen a wide range of presentations and edge cases. Ask whether they use FDA-approved formulations, compounded preparations, or both, and what the rationale is for each choice in a given patient. Ask about their approach to testosterone in women, including whether they routinely assess and treat low testosterone, and what monitoring protocols they use. Ask how they approach symptom management in women who are not candidates for systemic hormone therapy, including options like local vaginal estrogen, non-hormonal pharmacological treatments such as fezolinetant or paroxetine, and behavioral interventions supported by evidence [8].
Ask, specifically, what they think of the WHI study and how it informs their prescribing. A clinician who cites the original 2002 findings without nuance, or who uses them as a blanket justification for hormone therapy avoidance, has not followed the field. Ask how frequently they reassess hormone therapy decisions and what triggers a protocol change. The most revealing question is often the simplest: "What does a typical follow-up schedule look like for a patient on hormone therapy?" A specialist who monitors symptoms, blood levels, and cardiovascular markers at regular intervals is building a care relationship, not just writing a prescription.
Red Flags to Watch For
Several patterns of clinical behavior should prompt a patient to seek a second opinion. A clinician who refuses to discuss hormone therapy entirely, citing the WHI without engaging with subsequent evidence, is not operating from a current evidence base. The Menopause Society, the British Menopause Society, the International Menopause Society, and the Endocrine Society have all published position statements supporting the judicious use of hormone therapy in symptomatic women without absolute contraindications [8].
Conversely, a clinician who prescribes hormone therapy without any baseline laboratory work, without a discussion of individual risk, and without a monitoring plan is practicing with a degree of casualness that does not serve the patient. Custom-compounded hormone preparations promoted as universally superior to FDA-approved medications, without a clinical rationale specific to the individual patient, is a marketing claim, not a medical recommendation. Saliva testing marketed as the gold standard for hormone monitoring is another flag: it is not validated for clinical decision-making in the way that serum hormone measurements are [11].
A specialist who frames hormone therapy as a longevity intervention without acknowledging the genuine uncertainties that remain in long-term outcome data is overselling. A specialist who frames it as uniformly dangerous without engaging with the individualized risk-benefit framework is underselling. The right clinician holds both the evidence and the uncertainty with honesty, and communicates that honestly to patients.
How Telehealth Has Changed Access to Evidence-Based HRT
For most of the last two decades, finding a menopause specialist meant living in or near a major metropolitan area with academic medical center affiliations. Rural and suburban women faced a choice between a generalist who might be ambivalent about hormone prescribing and a specialist appointment months away. Telehealth has fundamentally disrupted that geography, and the disruption has been unambiguously positive for access to care.
The expansion of telehealth during and after the COVID-19 pandemic included regulatory changes that, in many jurisdictions, allowed physicians licensed in one state to consult patients in another, significantly broadening the available pool of prescribers for specialized conditions. Menopause medicine translates well to the telehealth model: most of the diagnostic work is laboratory-based, most monitoring is symptom-informed and lab-confirmed, and the clinical conversations that drive good outcomes require time and communication rather than physical examination [12].
Telehealth has not merely made menopause care more convenient. For women in rural areas, underserved communities, and states with limited specialist density, it has made specialist-level care possible for the first time.
The practical advantages extend beyond geography. Telehealth consultations tend to be longer and more focused than in-person visits in high-volume clinical settings. The absence of a waiting room, the ability to attend from home, and the asynchronous communication channels many telehealth platforms offer make it easier for patients to share symptom diaries, ask follow-up questions, and maintain the continuity that menopause management requires. Research on telehealth for chronic condition management consistently finds non-inferior outcomes compared to in-person care for conditions where laboratory monitoring and symptom tracking drive clinical decisions [12].
Hormone therapy dispensed through telehealth follows the same evidence-based frameworks as in-person care. Baseline labs are ordered through patient-convenient collection networks. Prescriptions are sent to licensed compounding or retail pharmacies with validated quality standards. Follow-up is scheduled at intervals appropriate to the protocol. The care model is different; the clinical standards are identical.
Integrating Menopause Care Into a Longevity Framework
The most forward-thinking menopause specialists situate hormone therapy not just as symptom management but as a component of a broader longevity strategy. This framing aligns with the trajectory of longevity medicine, which increasingly recognizes that the menopausal transition is one of the most biologically consequential inflection points in a woman's aging trajectory. The loss of estradiol accelerates several hallmarks of aging simultaneously: cellular senescence accumulates faster, mitochondrial function in skeletal muscle declines, neuroinflammation increases, and cardiovascular risk rises steeply [2].
Hormone therapy that is initiated at the right time, with the right formulations, and monitored appropriately is one intervention that addresses multiple aging pathways at once. It is not the only one, and a longitudinal approach to women's health in the menopausal years also includes attention to body composition, resistance exercise, sleep quality, metabolic health, and cardiovascular risk factor management. Women's hormone health programs that integrate all of these dimensions, like Women's Hormone Health, represent the clinical standard toward which menopause medicine is moving.
Metabolic health deserves particular attention during and after menopause. Estrogen plays an active role in insulin sensitivity and fat distribution, and its withdrawal shifts adipose tissue toward central, visceral deposition, which is more metabolically active and more inflammatory than subcutaneous fat. Insulin resistance tends to worsen in the postmenopausal years independent of weight change, meaning that metabolic monitoring should be a standard component of menopause care rather than an afterthought [5]. A Complete Female Hormone Panel combined with metabolic markers gives the clinical picture needed to personalize both hormone and lifestyle interventions effectively.
The intersection of menopause and bone health also connects directly to muscle health. Estrogen supports both bone maintenance and muscle protein synthesis. The perimenopause window, when estrogen begins fluctuating before it falls definitively, is an important period to establish resistance training habits and adequate protein intake, because these behaviors protect against the accelerated bone and muscle loss that follows the final menstrual period. A menopause specialist who addresses these domains together is practicing preventive medicine at its most impactful.
Perimenopause: The Overlooked Window
Perimenopause, the transition period that can begin four to ten years before the final menstrual period, is frequently missed or mismanaged in primary care. During this phase, estrogen does not simply decline: it fluctuates erratically, with periods of supraphysiological elevation alternating with sharp drops. These fluctuations drive symptoms including irregular periods, sleep disruption, mood instability, and changes in migraines or anxiety that are often attributed to depression, stress, or thyroid disease before the hormonal picture is considered [8].
A FSH measured on a single day during perimenopause may appear normal, because FSH fluctuates with the menstrual cycle and can be suppressed by one of those estrogen surges. Experienced menopause clinicians know that perimenopause is a clinical diagnosis supported by symptoms and context, not a laboratory diagnosis alone. A 44-year-old woman with classic perimenopausal symptoms and a "normal" FSH may still be in the transition and may benefit from hormonal support, including low-dose oral contraceptives for cycle regulation and symptom management, or progesterone to address sleep disruption and luteal phase insufficiency [8].
The failure to diagnose perimenopause correctly has real costs. Women who are misdiagnosed with depression during perimenopause and started on antidepressants that do not address the underlying hormonal fluctuation spend years on medications that are addressing a symptom, not a cause. Women whose sleep disruption is attributed to stress rather than nocturnal estrogen dips accumulate a sleep debt with measurable consequences for cardiovascular health, metabolic function, and cognitive performance. A menopause specialist who is alert to the perimenopausal window catches these women earlier, when intervention is most effective.
Practical Steps for Finding the Right Provider
The Menopause Society maintains a publicly searchable directory of Menopause Society Certified Menopause Practitioners at menopause.org, which is the most reliable starting point for finding credentialed providers. The directory is filterable by location and by whether the provider offers telehealth, which makes it a practical tool for women in underserved areas. The British Menopause Society and International Menopause Society maintain equivalent resources for patients outside North America.
Beyond directories, patient advocacy communities have become a significant source of provider intelligence. Menopause-focused online communities aggregate firsthand patient reports on provider quality, clinical philosophy, and prescribing approaches in ways that formal directories cannot capture. These reports should be weighed with appropriate skepticism, but patterns across many reports are often informative. A provider who consistently appears in patient communities as dismissive of symptoms or reflexively anti-hormone is revealing something about their clinical approach that a credential alone would not predict.
When evaluating a telehealth menopause program, the questions to ask are essentially identical to those for in-person care: what credentials does the prescribing clinician hold, what baseline assessment is conducted before prescribing, what monitoring protocol is in place, and what non-hormonal options are offered for women who cannot or choose not to use systemic hormones. A high-quality telehealth program will answer all of these questions clearly and be transparent about the evidence behind its clinical protocols.
The investment in finding the right menopause specialist is not a luxury. It is a form of preventive medicine with a decades-long return on investment, one that shapes cardiovascular aging, bone density, cognitive trajectory, body composition, and quality of life through the second half of a woman's life. The search may take more than one consultation and more than one provider before the right fit is found. That search is worth the effort.
Conclusion: The Search Is the Starting Point
The query "menopause specialist near me" begins as a geographic search but quickly becomes something more demanding: a search for a clinician who has kept pace with a rapidly evolving evidence base, who can navigate the legitimate nuances of hormone formulation and timing, who understands that menopause is an inflection point in long-term aging rather than a temporary symptom cluster, and who will build a care relationship calibrated to decades rather than months. That clinician exists. Credentials, clinical philosophy, and willingness to engage with current evidence are the filters that separate them from the rest of the field.
Telehealth has removed the geography barrier that previously made specialist-level menopause care inaccessible to millions of women. What remains is the work of identifying the right provider, asking the right questions, and insisting on an individualized, evidence-based approach rather than a standardized one. The biology of menopause does not negotiate: estrogen's withdrawal initiates changes in bone, brain, heart, and muscle that accumulate quietly over years before becoming clinically visible. The specialist who recognizes that timeline, and who acts within the early window when intervention is most consequential, is the one worth finding.
- Manson, J.E., Chlebowski, R.T., Stefanick, M.L., Aragaki, A.K., Rossouw, J.E., Prentice, R.L., ... & Wallace, R.B. (2009). Menopausal hormone therapy and health outcomes during the intervention and extended poststopping phases of the Women's Health Initiative randomized trials. Menopause, 16(3), 517–523. https://doi.org/10.1097/GME.0b013e3181cec27b
- Barton, M. (2018). Not just cardiovascular: the multidimensional actions of estrogens. Endocrine Reviews, 39(6), 869–893. https://doi.org/10.1210/er.2018-00151
- Maas, A.H., & Appelman, Y.E. (2010). Gender differences in coronary heart disease. Circulation, 136(1), e1–e9. https://doi.org/10.1161/CIR.0000000000000912
- Rizzoli, R., Branco, J., Brandi, M.L., Boonen, S., Bruyère, O., Cacoub, P., ... & Reginster, J.Y. (2013). Management of osteoporosis of the oldest old. Osteoporosis International, 25(11), 2507–2529. https://doi.org/10.1007/s00198-013-2465-4
- Maki, P.M., & Henderson, V.W. (2022). Cognition and the menopause transition. Maturitas, 163, 37–42. https://doi.org/10.1016/j.maturitas.2022.02.005
- The Menopause Society. (2024). Find a Menopause Practitioner Directory. https://www.menopause.org/for-women/find-a-menopause-practitioner
- Hodis, H.N., Mack, W.J., Henderson, V.W., Shoupe, D., Budoff, M.J., Hwang-Levine, J., ... & Selzer, R.H. (2016). Vascular effects of early versus late postmenopausal treatment with estradiol. JAMA, 315(24), 2674–2684. https://doi.org/10.1001/jama.2016.6456
- The Menopause Society. (2023). The 2023 Menopause Society position statement: hormone therapy. Menopause, 30(6), 613–666. https://doi.org/10.1097/GME.0000000000002200
- Lambrinoudaki, I., Brincat, M., Erel, C.T., Gambacciani, M., Moen, M.H., Schenck-Gustafsson, K., ... & Tremollieres, F. (2019). Progestogens in postmenopausal hormone therapy and the risk of breast cancer. Maturitas, 124, 91–96. https://doi.org/10.1016/j.maturitas.2019.04.007
- Davis, S.R., Baber, R., Panay, N., Bitzer, J., Perez, S.C., Islam, R.M., ... & Wierman, M.E. (2019). Global consensus position statement on the use of testosterone therapy for women. Journal of Sexual Medicine, 16(9), 1331–1337. https://doi.org/10.1016/j.jsxm.2019.03.012
- Stanczyk, F.Z., Cho, M.M., Endres, D.B., Morrison, J.L., Patel, S., & Paulson, R.J. (2012). Limitations of salivary estradiol measurements in postmenopausal women. Journal of Clinical Endocrinology & Metabolism, 97(8), 3571–3575. https://doi.org/10.1210/jc.2012-1861
- Snoswell, C.L., Taylor, M.L., Comans, T.A., Smith, A.C., Gray, L.C., & Caffery, L.J. (2021). Determining if telehealth can reduce health system costs: scoping review. Maturitas, 146, 74–82. https://doi.org/10.1016/j.maturitas.2021.04.008